Search results for "Growth hormone receptor"

showing 5 items of 5 documents

Circulating Irisin Levels in Children With GH Deficiency Before and After 1 Year of GH Treatment.

2018

Abstract Purpose To evaluate circulating irisin levels in children with GH deficiency (GHD) and any relation with clinical and metabolic parameters. Patients Fifty-four prepubertal children (mean age, 7.4 ± 0.8 years) with idiopathic GHD treated with GH for at least 12 months and 31 healthy short children as control subjects. Methods Body height, body mass index (BMI), waist circumference (WC), IGF-I, HbA1c, lipid profile, fasting and after–oral glucose tolerance test glucose and insulin, insulin sensitivity indices, and irisin levels were evaluated at baseline and after 12 months of GH replacement (GHR). Results At baseline, children with GHD, in addition to having lower growth velocity (P…

0301 basic medicineMalemedicine.medical_specialtyWaistHormone Replacement TherapyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical Biochemistry030209 endocrinology & metabolismGrowth hormone receptorBiochemistrySettore MED/13 - EndocrinologiaGrowth hormone deficiencyBody Mass Index03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceInternal medicinemedicineHumansProspective StudiesInsulin-Like Growth Factor IChildGrowth DisordersGlucose tolerance testmedicine.diagnostic_testbusiness.industryInsulinBiochemistry (medical)medicine.diseaseFibronectins030104 developmental biologyEndocrinologyTreatment OutcomeGrowth HormoneFemaleWaist CircumferenceLipid profilebusinessBody mass indexThe Journal of clinical endocrinology and metabolism
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Effect of circulating growth hormone on muscle IGF-I protein concentration in female mice with growth hormone receptor gene disruption.

2009

Growth hormone (GH) is a potent secretague for circulating insulin-like growth factor-I (IGF-I). The purpose of this study was to examine the effect of circulating GH on muscle IGF-I protein expression using GH transgenic animal models. Three different models were used: mice that overexpress bovine GH (bGH; n=10), mice without a functional GH receptor (GHR-/-; n=10), and wildtype mice (n=10). All mice were 16-week old females and each group differed in their basic phenotypic characteristics. Immediately after euthanization the triceps surae muscle group (soleus, plantaris, and gastrocnemius muscles) was removed. IGF-I was extracted from the muscle with an acid-ethanol solution (12.5% 2N hyd…

Malemedicine.medical_specialtyEndocrinology Diabetes and MetabolismTransgenemedicine.medical_treatmentMice TransgenicGrowth hormone receptorBiologyMiceEndocrinologyTriceps surae muscleInternal medicinemedicineAnimalsInsulin-Like Growth Factor IReceptorMuscle SkeletalInsulin-like growth factor 1 receptorMice KnockoutGrowth factorRadioimmunoassayReceptors SomatotropinMice Inbred C57BLEndocrinologyGrowth HormoneCattleFemaleHormoneGrowth hormoneIGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
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Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population

2014

SummaryMice and humans with growth hormone receptor/IGF-1 deficiencies display major reductions in age-related diseases. Because protein restriction reduces GHR-IGF-1 activity, we examined links between protein intake and mortality. Respondents aged 50–65 reporting high protein intake had a 75% increase in overall mortality and a 4-fold increase in cancer death risk during the following 18 years. These associations were either abolished or attenuated if the proteins were plant derived. Conversely, high protein intake was associated with reduced cancer and overall mortality in respondents over 65, but a 5-fold increase in diabetes mortality across all ages. Mouse studies confirmed the effect…

Malemedicine.medical_specialtyLow proteinnutrition protein intake caloric restriction nutrientsPhysiologymedicine.medical_treatmentLongevityCalorie restrictionBreast NeoplasmsGrowth hormone receptorBiologyArticleMiceLow-protein dietNeoplasmsDiabetes mellitusInternal medicineDiabetes MellitusDiet Protein-RestrictedmedicineAnimalsHumansInsulin-Like Growth Factor IMelanomaMolecular BiologyAgedProportional Hazards ModelsMice KnockoutMice Inbred BALB CIncidence (epidemiology)CancerCell BiologyMiddle Agedmedicine.diseaseMiddle ageMice Inbred C57BLCross-Sectional StudiesEndocrinologyFemaleCarrier ProteinsFollow-Up StudiesSignal TransductionCell Metabolism
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Janus kinase (JAK) 2 V617F mutation as the cause of primary thrombocythemia in acromegaly with severe visceromegaly and divergence between growth hor…

2012

OBJECTIVE: An increased prevalence of hematological abnormalities is reported in acromegaly, but to date no reports about the presence of the Janus Kinase (JAK) 2 mutation in acromegalic patients have been described. DESIGN: We report the complex clinical presentation of the unique case, never described, of acromegaly due to GH-secreting pituitary adenoma associated with JAK2 V617F mutation. RESULTS: The patient shows primary thrombocythemia and myelofibrosis, due to JAK2 V617F mutation, severe visceromegaly and a peculiar clinical course of the disease characterized by discrepant values of GH and IGF-1 during somatostatin analog (SA) treatment despite a significant reduction in pituitary a…

Malemedicine.medical_specialtyTime FactorsEndocrinology Diabetes and MetabolismGrowth hormone receptorAcromegaly HGH IGF-1GastroenterologySettore MED/13 - EndocrinologiaCohort StudiesEndocrinologyPituitary adenomahemic and lymphatic diseasesInternal medicineAcromegalymedicineAnimalsHumansPituitary NeoplasmsInsulin-Like Growth Factor IRadiometryMyelofibrosisAgedJanus kinase 2biologyHuman Growth HormonePlatelet Countbusiness.industryJanus Kinase 2medicine.diseaseAcromegaly Growth hormone Insulin-like growth factor-1 Janus kinase 2 Growth hormone receptorEndocrinologyAcromegalyMutationPegvisomantbiology.proteinJanus kinasebusinessVisceromegalyFollow-Up StudiesThrombocythemia Essentialmedicine.drug
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Growth hormone protects human lymphocytes from irradiation-induced cell death

2003

1. Undesired effects of cancer radiotherapy mainly affect the hematopoietic system. Growth hormone (GH) participates in both hematopoiesis and modulation of the immune response. We report both r-hGH cell death prevention and restoration of secretory capacities of irradiated human peripheral blood lymphocytes (PBL) in vitro. 2. r-hGH induced cell survival and increased proliferation of irradiated cells. Western blot analysis indicated that these effects of GH were paralleled by increased expression of the antiapoptotic protein Bcl-2. 3. r-hGH restored mitogen-stimulated release of IL-2 by PBL. Preincubation of irradiated lymphocytes with the growth hormone receptor (GHR) antagonists B2036 an…

Pharmacologymedicine.medical_specialtyProgrammed cell deathmedicine.diagnostic_testCellCancerGrowth hormone receptorBiologymedicine.diseaseHaematopoiesisEndocrinologymedicine.anatomical_structureImmune systemWestern blotApoptosisInternal medicinemedicineCancer researchhormones hormone substitutes and hormone antagonistsBritish Journal of Pharmacology
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